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목적: 성장성 두개골 골절은 영아나 소아에서 두경부 손상후에 발생한다. 저자들은 성장성 두개골 골절로 진단된 환아 들의 MR 소견을 보고자 한다. 대상과 방법: 성장성 두개골 골절로 진단된 5명의 MR 영상을 후향적으로 분석하였고 남녀 비는 2:3 이었으며 평균연령 은 7.5세 였다. 외상의 과거력과 성장성 두개골 골절로 내원 할 때까지 기간은 3달에서 12년까지 넓게 분포하였다. 단 순 촬영과 CT가 있는 환아에서는 이들 영상 소견을 같이 분석하였다. 결과: 성장성 두개골 골절의 가장 흔한 위치는 두정엽 부위였으며 MR 에서 외상후 실질 변화로 낭성 뇌연화증과 공뇌 증이 보였다. 골절 변연부 골 비후와 판사이 공간 확장이 4예에서 보였다. 이러한 모든 병변을 보여주는데 있어 MR 영 상은 매우 우수하였다. 결론: 성장성 두개골 골절 환아에서 MR 영상은 외상 후의 뇌 실질 변화와 골절 변연부 골비후와 같은 골 변형 및 재형성 등을 분명하게 보여 주었다.


Purpose: Leptomeningeal cyst or growing skull fracture can occur in young infants or children following head trauma. We present MR imaging findings in five children with growing skull fracture. Materials and Methods:We reviewed the MR images of five children (M: F=2:3) with growing skull fracture. The mean age was 7.5 years. The time interval between the occurrence of head trauma and the presentation of growing skull fracture varied from three months to 12 years. We reviewed the precontrast CT scans and/or the plain skull radiographs in those patients for whom these studies were available. Results: The most common location of the growing skull fracture was the parietal bone (n=3). On the MR images, there were bone defects with posttraumatic cystic encephalomalacia or porencephalic cysts. Marginal bony thickening and diploic space widening were noted in four patients. MR imaging was excellent for visualizing the parenchymal changes and pericranial lesions. Conclusion: In children with growing skull fracture, MR imaging can clearly depict trauma-related parenchymal changes, pericerebral lesions as well as bony edge thickening with remodeling.


Purpose: Leptomeningeal cyst or growing skull fracture can occur in young infants or children following head trauma. We present MR imaging findings in five children with growing skull fracture. Materials and Methods:We reviewed the MR images of five children (M: F=2:3) with growing skull fracture. The mean age was 7.5 years. The time interval between the occurrence of head trauma and the presentation of growing skull fracture varied from three months to 12 years. We reviewed the precontrast CT scans and/or the plain skull radiographs in those patients for whom these studies were available. Results: The most common location of the growing skull fracture was the parietal bone (n=3). On the MR images, there were bone defects with posttraumatic cystic encephalomalacia or porencephalic cysts. Marginal bony thickening and diploic space widening were noted in four patients. MR imaging was excellent for visualizing the parenchymal changes and pericranial lesions. Conclusion: In children with growing skull fracture, MR imaging can clearly depict trauma-related parenchymal changes, pericerebral lesions as well as bony edge thickening with remodeling.